@article {10.3844/ajptsp.2026.16.20,
article_type = {journal},
title = {Alcohol-Induced Rhabdomyolysis Complicated by Hyponatremia and Acute Kidney Injury Following Minor Trauma: A Case Report},
author = {Tulsian, Vinay and Das, Drupad and Sukumar, M and Panda, Prasan Kumar},
volume = {20},
year = {2026},
month = {May},
pages = {16-20},
doi = {10.3844/ajptsp.2026.16.20},
url = {https://thescipub.com/abstract/ajptsp.2026.16.20},
abstract = {Rhabdomyolysis from alcohol is established, but combined binge intake, minor trauma, and vomiting-induced hyponatremia is a complex clinical scenario. Case study: This case report describes a young male with a three-year history of alcohol consumption who presented with rhabdomyolysis following binge drinking and a mild traumatic fall. He experienced 10 to 15 episodes of vomiting post-alcohol intake, subsequently developing anasarca. He reported decreased urine output and right-hand weakness. Examination revealed right-sided ulnar nerve involvement, and Clinical evaluation revealed acute kidney injury (creatinine: 8.44 mg/dL) and rhabdomyolysis (Creatine phosphokinase: 8872 U/L), attributed to a combination of trauma, excessive alcohol intake, and hyponatremia (serum sodium: 118 mmol/L) resulting from vomiting. Imaging studies ruled out bony injuries but identified an organized hematoma in the right gluteus maximus and myositis in bilateral thighs. Management and outcome: the patient received aggressive hydration and corrective measures for severe hyponatremia. By day 10 of hospitalization, his condition improved significantly, and during Follow-up visits, he showed complete resolution. Conclusion: This case illustrates how multiple synergistic triggers can precipitate severe rhabdomyolysis and underscores the importance of prompt and comprehensive intervention for successful patient outcomes.},
journal = {American Journal of Pharmacology and Toxicology},
publisher = {Science Publications}
}